South Baylo University - School of Acupuncture and Oriental Medicine

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Chronic Pain Control Center

Jun's M-puncture - Introduction

Molecular lesions that were created in the C-fiber and its central pathway are the axial cause of chronic persistent pain.
M-puncture is targets molecular lesions in C-Fiber and its central pathway by Aδ fiber stimulation through the damaging stimuli with PML Targeting.

Based on recent outstanding molecular research of pronociceptive processing in C-fiber and central nociceptive neuron since 1999, neuronal plasticity has pivotal roles in inducing chronic pain from acute pain and further maintaining chronic pain with implication of glial system and peripheral immune system.
Therapeutic targets for control of chronic pain have to be changed from nociceptive lesion (trigger points, arthritis, strain, inflammation, injury, etc.) to molecules that make neuronal plasticity in C-fiber and central nociceptive neurons.
Principle mechanism of medical acupuncture is based on using the endogenous analgesic system which suppresses pain transmission from nociceptive lesions to several cortex by supraspinal and contralateral mediation and then from supraspinal to dorsal horn (e.g. DNIC).

M-puncture sites are not modified auriculoacupuncture points.

Inverted projection of embryo (Nogier,1956)
Modified & developed in China
Embryology
Still effectively used in TCM
Point locations mainly in scapha (for shoulder pain) and antihelix (for musculoskeletal pain)

Chronic pain due to molecular lesions and neuronal plasticity in C-fiber pathway(1999)
Targets V3, C2, C3, X stimulation
Completely distinct point locations from auriculoacupuncture
Point locations mainly in fossa triangularis and concha
M-puncture is unrelated to embryological origin

V1 stimulation
Eight zones on frontal portion of scalp
Point location not based on affected brain regions
Targets molecular lesions in C-fiber and its nociceptive neuronal system pathway

1. Medical Acupuncture

Pain inhibit pain concept = Pain suppressive system activation by needling

(Usage = Pain stimulation (pain lesional side) → DH
Contralateral ascending spinal cord and brain stem nuclei (contralateral) → Brain stem nuclei (pain lesional side) → DH (pain lesional side) to pain suppression.
So axial mechanism of medical acupuncture is endogenous analgesic system with segmental stimulation

2. M-puncture - Molecular targeting concept targets at neuronal plasticity lesions which initiate and increase

the nociception of neuron itself, resulting in chronic pain (After 1999 Molecular mechanism in

Pain processing (RR Ji, CJ Woolf))

Mainly through ipsilateral targeting concept for relief and molecular lesions using ipsilateral DI and ipsilateral PAF and DH (PML, SML, Activation of ipsilateral DI and suppression of ipsilateral PAF). This is the point of the difference between EA and M-puncture
Traditional EA effect add to A